To digress, as I mentioned in the first blog post of this series the reason, I have not blogged about vitamin D3 and its direct role in destroying/preventing cancer, is because it was not clear to me how vitamin D3 was involved. It was only recently after more research on Read more…
Taking skin cells as an example in skin cells-keratinocytes-all three main forms of vitamin D3 activate the HPA axis and the peripheral cellular process where this occurs to produce pro-opiomelanocortin (POMC). 1 To summarize the three main forms of vitamin D3. Cholecalciferol- the form of vitamin D3 produced from UV-B Read more…
Sunscreens as I previously blogged about are not only poisoning both us as well as our environment but affect the p53, one of our main ways to fight cancer. Particularly skin cancers. Less sun by whatever means, results in less pro-opiomelanocortin (POMC). And thus less melanotropin stimulating hormone (MSH) produced Read more…
To digress a minute let me talk about the P53 gene and P53 protein this gene produces as both are important in cancer suppression and progression. The P53 gene is called the cancer suppressor gene. P53 is known for both slowing the cell replication cycle and destroying cells. By slowing Read more…
The blood storage for of vitamin D3 is activated by 1-alpha hydroxylase enzyme into the active form of vitamin D3 (1,25 dihydroxy vitamin D3). Once activated it must still be modified by proteins to once it interacts with the vitamin D receptor (VDR) to initiate activity of gene(s). Concerning the Read more…